Inflammation, mitochondria, and the role of ubiquinol in recharging cardiac energy

Cardiovascular disease (CVD) remains the leading cause of morbidity and mortality worldwide,¹ yet our perspectives of its pathophysiology continue to expand. Beyond cholesterol accumulation and vascular obstruction, growing evidence implicates impaired energy metabolism and chronic inflammation, both closely linked to mitochondrial health with CVD.² Mitochondria produce adenosine triphosphate (ATP) to sustain the constant, high-energy demands of the myocardium - when mitochondrial efficiency falters, cardiac performance deteriorates.³